Familial acute leukemia with mutant CEBPA, is one form of cancer of blood-forming tissue (bone marrow) called acute leukemia. In normal bone marrow, early blood cells called necrosis stem cells turn into several types of blood cells: white blood cells (leukocytes), which protect the body from infection, red blood cells (erythrocytes) that have oxygen and blood platelets (points), which are involved in the clotting of blood. Acute leukemia bone marrow produces a large number of abnormal white blood cells, immature, blasts called szpikowa. Instead of developing to a normal white blood cells, szpikowa blasts develop into bronchial cells leukemia. A large number of abnormal cells in the bone marrow interferes with the production of white blood cells, functional red blood cells and blood platelets.
People with familial acute leukemia with CEBPA mutated to a shortage of white blood cells (leukopenia), leading to increased susceptibility to infections. A low number of red blood cells (anemia) also occurs in this disorder, causing fatigue and weakness. People affected by the disease are also reducing the amount of blood platelets (Thrombocytopenia), which may result in easy bruising and irregular bleeding. Other symptoms of familial acute leukemia with mutant CEBPA may include the loss of the horse and weight.
Acute leukemia in General is a disease of older people, familial acute leukemia with mutant CEBPA often starts earlier in life and has been reported to occur already at the age of 4. Between 50 and 65 per cent of the persons affected to survive their disease, compared to 25 to 40 per cent of these other forms of acute leukemia. However, people with familial acute leukemia with CEBPA mutant have higher risk of a new instance of the core is the disorder after successful treatment of the initial instance.
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